Preliminary experiments suggest conolidine may well inhibit distinct ion channels, reducing neuronal excitability and restricting suffering signals. This mechanism is especially relevant in neuropathic suffering, where by irregular signaling triggers persistent irritation. Also, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to agony notion. Computat... https://confuciusa173owg9.wikijournalist.com/user