Preliminary studies point out conolidine may inhibit particular ion channels, lessening neuronal excitability and limiting soreness alerts. This mechanism is especially suitable in neuropathic agony, where abnormal signaling triggers persistent soreness. Furthermore, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to soreness perception. Computational ... https://sandrae173xqh0.wikipowell.com/user